ITGB2 and familial hyperaldosteronism: Similarly to neutrophils, FH was shown to influence a wide plethora of monocyte and macrophage functions too, such as chemotaxis (24), stimulation of respiratory burst (25) and cytokine secretion (26), mainly via the complement receptors CR3 (CD11b/CD18) and CR4 (CD11c/CD18) (14, 27).