The different biodistributions of [68Ga]-Ga-PSMA-11 and [18F]-F-PSMA-1007 led to disconcordant tracer accumulations, especially in lesions of unspecific/benign origin and to a far lesser extent in prostate cancer manifestations, which is in line with the previously published head-to-head [17] and matched comparisons [16]. This evidence concerns the gene FOLH1 and prostate carcinoma.