We next examined expression of CCL2/MCP-1, a type I IFN–stimulated cytokine implicated in monocyte chemotaxis (75) and up-regulated in lung samples from patients with COVID-19 (39, 40), as well as MX1, an antiviral response gene induced by type I/III IFNs and up-regulated during SARS-CoV-2 infection (76). This evidence concerns the gene CCL2 and COVID-19.