In comparison, in the control H1 human embryonic stem cell line, only 15 of 17,000 KMT2A peaks were called as wide and these peaks were significantly less enriched on gene TSSs than the wide KMT2A peaks we identified in CD34+ HSPCs and significantly less enriched in gene bodies than the oncoprotein-binding sites in KMT2Ar leukemia samples (Fig. 1e and Extended Data Fig. 2m). This evidence concerns the gene CD34 and leukemia.