CD8A and gastric cancer: The previous report showed that Treg from GC could decompose ATP into adenosine, then induced apoptosis and inhibited the proliferation of CD8 + T cells through A2AR pathway, which further leads to immune escape of GC63.To sum up, combined with the results of this study, the crosstalk between miR-30c-5p target gene expression and some immune cells could promote the development of gastric cancer.