TFF2 and ancylostomiasis: Although our work in mouse models of hookworm infection suggests that both TFF2 and TFF3 contribute to worm clearance, tissue repair, and the suppression of proinflammatory cytokines [32–35], we found that TFF2 is specifically increased in the context of human hookworm infection and can reduce secretion of proinflammatory cytokine by human PMBCs.