At the same time, the miR-203/SNAI2 axis emerged as a high-profile symbol in tumor stemness, EMT, and angiogenesis in prostate cancer, in which suppression of miR-203 on transcriptional inhibitor SNAI2 is relieved due to lessened miR-203 existence, rendering reanimation of the downstream oncogenic GSK-3β/β-CATENIN signal pathway by activated SNAI2 [82]. Here, SNAI2 is linked to neoplasm.