In reference to neuropathological characteristics of anti-IgLON5 disease, in which subcortical pTau deposits were found, with isolated tau pathology in the temporal cortex only, this could also explain the presence of SWJ in patients with PSP in which tau pathology is found more prominently over the course of the disease (4, 23, 26). This evidence concerns the gene IGLON5 and supranuclear palsy, progressive, 1.