And we have proved that Gal-9 high-expressing ERCs can further reduce the progression of ConA-induced experimental autoimmune hepatitis, by reducing the infiltration of CD4+ and CD8+ T cells in the liver, suppressing Th1 and Th17 cell response, inhibiting CD4+ and CD8+ T cell proliferation in the hepatitis mice, and regulating inflammatory factors. Here, LGALS9 is linked to Hepatitis.