This perhaps explains the greater magnitude of increase in NF-κB2 mRNA found when all people with schizophrenia are grouped together [11] as compared to RelA and NF-κB1 mRNA increases that would be less, on average, considering that non-inflamed people with schizophrenia make about 60% of the cases [50–52]. The gene discussed is NFKB2; the disease is schizophrenia.