Not only does it inhibit a multitude of pathways that were upregulated in NPC tissue, such as IL-1B and COX-2, it may also modulate IFN-G (as reported in the literature and in our analysis) and Akt-mediated signaling, both of which are related to poor prognosis in NPC [74]. The gene discussed is IFNG; the disease is nasopharyngeal carcinoma.