Among them, we found Glioma pathogenesis-related protein 1 (glipr1), Receptor of activated protein C kinase 1 (rack1 or gnb2l1), Protein disulfide-isomerase (p4hb), and Carbonic anhydrase 9 (ca9) known to be over-expressed in glioblastoma and to promote tumor growth (41–44). Here, GLIPR1 is linked to neoplasm.