Next, the expression levels of the pro‐inflammatory cytokines (IL‐1β and IL‐18) were examined by conducting Real‐Time qPCR and ELISA analysis in AR42J cells and its supernatants, and our findings indicated that upregulation of tRF3‐Thr‐AGT attenuated STC‐induced cellular inflammation in AR42J cells (Figure 4E–H), implying that tRF3‐Thr‐AGT participated in the regulation of cell pyroptosis and inflammation during AP progression in vitro. The gene discussed is IL18; the disease is alkaline phosphatase measurement.