In our associate publication [18] we focus on the computational studies of the predicted molecular interactions between arginine, a putative pharmacochaperone that was found able to improve the activity of p.Gln188Arg and other GALT mutants in a prokaryotic model of classic galactosemia [19], and both the wild-type and mutant p.Gln188Arg enzymes. Here, GALT is linked to classic galactosemia.