Furthermore, Yang et al. [106] reported that the interaction between ESC and Mφ could decrease the cytotoxicity of natural killer (NK) cells, possibly stimulating the secretion of IL-10 and TGF-β by Mφ, and in this way, they could favor the immunological escape of ectopic fragments and promote the appearance and development of endometriosis. Here, IL10 is linked to endometriosis.