In the case of the model built with the G. Haemolysan abundance and tumor presence variables, it tells that the estimated increase in CCL2 associated to G. haemolysans is higher than to the other bacteria and, contrary to F. Nucleatum and B. fragilis, the increase in CCL2 levels is totally influenced and explained by the increase in bacterium abundance, as their association is stronger, making the presence of tumor irrelevant. Here, CCL2 is linked to neoplasm.