Moreover, the anti-inflammatory effects of oxytocin treatment have been shown also in a rat model of myocardial infarction, where oxytocin reduced cell infiltration, such as neutrophils, macrophages, and T-lymphocytes in infarcted cardiac areas; interestingly, such effect was accompanied by a reduction in the expression of proinflammatory cytokines, such as TNF-α, IL-6, and IL-1β, and stimulation of the anti-inflammatory cytokine TGF-β [122]. The gene discussed is OXT; the disease is myocardial infarction.