Concurrently, sepsis caused higher gene sets enrichment in PCV in the following GO terms: cell chemotaxis, cell–cell adhesion by cadherins, cell–cell adhesion by integrins, chemokine biosynthesis process, calcium-mediated signaling, negative regulation of blood circulation, negative regulation of plasminogen activation, negative regulation of sodium ion transport, regulation of actin filament process, neutrophil homeostasis and response to prostaglandin (Figure 4A). This evidence concerns the gene CDH17 and Sepsis.