Here, we identified that OCM remarkably enhanced FASN and CPT1A expression in ES-2 cells compared with coculture in 1% FBS-negative control medium, whereas the OCM-mediated upregulation of FASN and CPT1A was attenuated upon overexpression of miR-33b in ovarian cancer cells (Figure 5A). This evidence concerns the gene CPT1A and ovarian carcinoma.