U2AF2 and infection: Given that the IWS1 phosphorylation-dependentalternative RNA splicing of U2AF2 regulatesthe nuclear export and translation of type I IFN genes, we hypothesized thatcells in which IWS1 was knocked down and cells in which the IWS1 knockdown wasrescued with the phosphorylation site mutant of IWS1 will be more sensitive toviral infection.