Furthermore, we have recently demonstrated that several STE20-type kinases – STK25, MST3, and MST4 – associate with intrahepatocellular lipid droplets and critically orchestrate liver lipid partitioning and NAFLD development [[9], [10], [11], [12], [13], [14], [15], [16], [17]]. This evidence concerns the gene STK26 and metabolic dysfunction-associated steatotic liver disease.