GATA2 and myelodysplastic syndrome: Data collected from FACS analyses showed that heterozygous GATA2 mutations (R396Q and R398W) lead to a block of early hematopoietic progenitor maturation (CD34+CD43+CD45+) and to an augment of myeloid progenitors’ compartment (CD33+/CD14+), which is in line with a low-risk MDS stage in childhood.