In accordance with previous studies,16 we confirmed that H37Rv infection in THP‐1‐derived macrophages induced increased TNF‐a expression and indicated the M1 polarization potential; compared with BCG or H37Ra infection, H37Rv infection selectively induced IL‐23 rather than IL‐12 in THP‐1‐derived macrophages. This evidence concerns the gene TNF and infection.