Further KEGG analysis showed that high‐risk groups also enriched in several pathways and processes, which were closely related to tumour progression, including adhesion junctions, ECM receptor interactions, O‐glycan biosynthesis, steroid biosynthesis, TGF‐β signaling pathways and ubiquitin‐mediated proteolysis (Figure 6B). The gene discussed is TGFB1; the disease is neoplasm.