NFAT5 and intracerebral hemorrhage: A previous study found that BMSC-Exos rich in miR-146a-5p can directly down-regulate the expression of IRAK1 and NFAT5, inhibit the polarization of microglia M1 after intracerebral hemorrhage, reduce the inflammatory response, and reduce nerve cell apoptosis in male rats with intracerebral hemorrhage, and in turn protect and improve nerve function after cerebral hemorrhage (Duan et al., 2020).