MCAO induced NF-κB activation, as manifested by an increase in both the percentage of phos-IκBα (Ser32) in IκBα (Figures 8(a) and 8(e)) and percentage of phos-p65 (Ser536) in p65 (Figures 8(a) and 8(h)), 21 days after stroke. Here, NFKB1 is linked to stroke disorder.