Dihydroartemisinin (DHA) inhibits lung cancer cell proliferation and colony formation, enhances cell death, and induces ferroptosis by inactivating the polypeptide 2 (PRIM2)/SLC7A11 axis, indicating that the inhibitory expression of PRIM2 may be a potential therapeutic target for lung cancer [177]. Here, SLC7A11 is linked to lung carcinoma.