CD8A and myocardial infarction: 1Adoptive transfer of Tregs attenuated cardiac remodeling by reducing IFN-γ expression in MI (98).2Exosomes produced by Tregs inhibited the proliferation and cytokine secretion of Th1 cells that may be involved in the regulation of heart damage (99).3Tregs inhibited the response of CD8+ T cells after MI by reducing pro-inflammatory factors of CD8+ T cells (22).4Tregs from HF patients were dysfunctional in suppressing the function of responder T cell (Tresp) (39).