Though we did not assess PKC in this study (which is one of the study’s limitation), however, the observed increase in cell viability by BC that was accompanied with corresponding decrease in caspase 3 activity and maintenance of ∆Ψm is suggestive of the fact that BC could have similarly used PKC as a downstream signalling molecule for its anti-apoptotic effect. This evidence concerns the gene PRRT2 and breast cancer.