NR4A1 and Burkitt lymphoma: Subsequent studies demonstrated that in BL cell lines, BCR-induced rapid activation of CaN resulted in the nuclear translocation of NFATc2, a member of the family of nuclear factor of activated T-cells (NFAT) transcription factors, followed by the increased expression of pro‐apoptotic molecule TR3/Nur77 and initiation of the apoptotic programme [57].