FOXP4 and neoplasm: Cai et al demonstrated that simultaneous inhibition of FGFR and mTOR activity contributed to anti-proliferative effects and tumor regression in ovarian cancer.[32] The JAK/STAT signaling pathway plays a vital role not only in the transformation of stationary epithelial cells to invasive and migratory cells but also in the maintenance of stem cell self-renewal.[42] Therefore, we speculate that FOXP4-AS1 plays an important role as an anticancer gene inhibiting the transformation of ovarian epithelial cells into invasive and migratory cells through this pathway.