Cai et al demonstrated that simultaneous inhibition of FGFR and mTOR activity contributed to anti-proliferative effects and tumor regression in ovarian cancer.[32] The JAK/STAT signaling pathway plays a vital role not only in the transformation of stationary epithelial cells to invasive and migratory cells but also in the maintenance of stem cell self-renewal.[42] Therefore, we speculate that FOXP4-AS1 plays an important role as an anticancer gene inhibiting the transformation of ovarian epithelial cells into invasive and migratory cells through this pathway. This evidence concerns the gene SOAT1 and neoplasm.