However, from the in vitro cell proliferation and lipidomic analysis experiments, it was found that the in vitro proliferation of glioma cells and oncogenic ether ester expression changes caused by AGPS silencing could not be completely reversed by HNRNPK rescue, so it is possible that AGPS still exists other target genes in gliomas and needs to be explored. This evidence concerns the gene HNRNPK and central nervous system cancer.