Through the HNRNPK rescue experiment, after upregulating the expression of HNRNPK in the AGPS-silenced group cells, the proliferation ability of the cells in vitro and the cancer-promoting lipid content can be partially restored, further confirming that HNRNPK plays an important role in AGPS in regulating the proliferation and lipid synthesis of glioma. The gene discussed is HNRNPK; the disease is cancer.