HNRNPK and central nervous system cancer: Through the HNRNPK rescue experiment, after upregulating the expression of HNRNPK in the AGPS-silenced group cells, the proliferation ability of the cells in vitro and the cancer-promoting lipid content can be partially restored, further confirming that HNRNPK plays an important role in AGPS in regulating the proliferation and lipid synthesis of glioma.