Ackermann et al. (30) also found evidence of the following features in a cohort of 7 COVID-19 post-mortem lung samples: diffuse alveolar damage; edema; angiotensin-converting enzyme 2 (ACE2) expression in epithelial and endothelial cells; endothelial damage and viral infection; and T-cell infiltration. Here, ACE2 is linked to COVID-19.