Immunofluorescence (IF) analyses of U87 and T98G human GBM cells with an anti-GLUT1 antibody and co-staining the cells with cholera toxin subunit B (CTB) conjugated with Alexa Fluor 594, a tracer for the PM, showed that 2-BP treatment induced redistribution of GLUT1 protein from the PM to the cytoplasm (Fig. 1a). Here, SLC2A1 is linked to glioblastoma.