Similarly, gene expression of tight junction proteins (ZO-1, Occludin, JAM-A, or Claudin 1) or inflammatory markers (IFN-γ, IL-1, and TNF-α) were not significantly different between mast cell–deficient KitW/W–v mice randomized to RC versus HFM groups (Supplemental Figure 2). This evidence concerns the gene IL1B and craniofacial microsomia.