CXCR3, which mediates chemotaxis in response to IFN-induced inflammatory chemokines (CXCL9, CXCL10, CXCL11), was also upregulated in COVID-19 patients’ DC3, cDC2 and monocyte subsets but downregulated in cDC1, tDC, and pDC. This evidence concerns the gene CXCR3 and COVID-19.