Remarkably, we found higher expression of CD86 and HLADR and lower expression of PD-L1 in the Ki67+ fractions of cDC2 and DC3 (Fig 5F and 5G), suggesting that the PD-L1hi CD86lo phenotype in DCs of COVID-19 patients was not caused by the recruitment of immature progenitors from the bone marrow, but could have been induced by external factors such as inflammatory mediators in the blood. The gene discussed is MKI67; the disease is COVID-19.