Respiratory damage is the most fatal complication of COVID‐19 patients; the coronaviruses invade nasal epithelium through the high co‐expression levels of angiotensin‐converting‐enzyme 2 (ACE2) and transmembrane protease serine 2 (TMPRSS2), as transmembrane SARS‐CoV 2 receptors, it uses nasal epithelium not only for penetration but also as the main target.3 Here, TMPRSS2 is linked to COVID-19.