The role of TIM-1 in the regulation of immune reactions in a human is indirectly confirmed by the fact that genetic TIM-1 polymorphisms in some ethnic populations are associated with bronchial asthma [37–40] and systemic lupus erythematosus [41], correlate with the inflammatory reaction intensity, and the severity of community-acquired pneumonia in children [42]. The gene discussed is HAVCR1; the disease is systemic lupus erythematosus.