Besides, in healthy controls, we obtained inactivated pathways related to viral responses (herpes, hepatitis B, hepatitis C, measles, Epstein-Barr virus), antiviral pathways (IFN-1 production), and inflammatory-related pathways (C-type lectin receptor signaling pathway, NOD-like receptor signaling pathway, Th17 cell differentiation, and inflammation guided by chemokine and cytokine signaling; Figure 14B). This evidence concerns the gene CLEC4D and hepatitis B virus infection.