Our findings show that Ang II infusion increased LFSBP, a marker of sympathetic vasomotor modulation (deBoer et al., 1987; Madwed et al., 1989), which is consistent with reports in other rodent models of HTN (Waki et al., 2006; Zubcevic et al., 2014, 2017; Chaar et al., 2016). The gene discussed is AGT; the disease is hypertensive disorder.