Here, we showed that several tumors (including THYM, OV, LIHC, HNSC, BLCA and LUAD) with SWI/SNF CNV alterations demonstrated significantly higher levels of TMB (Fig. 3A) and tumor neoantigen burden (TNB, Fig. 3B) than the subtypes without CNAs (P < 0.05). This evidence concerns the gene SMARCA1 and neoplasm.