NCR3 and neoplasm: Given the selective NKp30 upregulation in SG tissue of pSS and the NCR evolvement in tumor surveillance, we investigated whether the NKp30 expression on circulating NK cells (expressed both as frequency and MFI) was different according to the presence of SG inflammatory infiltration (regardless of the ELS organization), myoepithelial sialadenitis (MESA) pre-lymphomatous lesions, or non-Hodgkin’s MALT lymphoma in SS parotid SG.