For instance, in myocardial damage mediated by sepsis, the competitive binding of KCNQ1 opposite strand/antisense transcript 1 (KCNQ1OT1) and miR-192-5p modulates the X-linked inhibitor of apoptosis (XIAP) expression, and KCNQ1OT1 overexpression mitigates myocardial injury stemming from sepsis [7]. The gene discussed is KCNQ1OT1; the disease is Sepsis.