In addition, although SQ-LNSs substantially reduced the prevalence of IDA, they did not completely eliminate IDA, possibly reflecting factors such as poor iron absorption (due to antinutritional compounds, gastric pH, or intestinal or systemic inflammation) (1) or an Hb cutoff for anemia that is set inappropriately high for infants and young children, especially in African populations (55–57). The gene discussed is GSTM1; the disease is anemia.