STAT3 and hepatocellular carcinoma: In addition, there was an obvious increase in p‐C‐MET and p‐STAT3 levels in PLAGL2‐overexpressing Bel‐7402 cells compared to those in HGF‐stimulated Ctrl/Bel‐7402 cells (Figures 3J, and S4D), indicating that C‐MET‐regulated signaling contributed to PLAGL2‐induced HCC apoptosis resistance.