Bone morphogenetic proteins (BMPs), inorganic pyrophosphate, as well as factors facilitating calcification such as fetuin-A (alpha 2-Heremans–Schmid glycoprotein), matrix-Gla protein (MGP), osteoprotegerin (OPG) levels of calcification inhibitors and also the deterioration of the balance between these factors plays an important role in the increase of vascular calcification observed in uremia and atherosclerosis [6–9]. Here, MGP is linked to atherosclerosis.