Gel- analysis of the present study revealed that 103 out of 139 msp-1 amplicon (74%) were monoclonal infections, whereas the remaining 36 (26%) was poly-allelic type, with 15% for (MAD20 + K-1), 5.7% for (MAD20 + RO33), 2.8% for (K-1 + RO33), and 2.1% were MAD20 + K-1 + RO33 type. This evidence concerns the gene ATAD1 and infection.