The results of our study indicated that GBBC may have a strong anti-inflammatory effect in the mouse model of L-arginine-induced AP, as evidenced by decreased MPO activity (in the pancreas and lungs) and selected biochemical markers (in blood serum), as well as—most notably—histological abnormalities in the pancreatic tissue (H&E staining, Masson’s trichrome staining). Here, MPO is linked to alkaline phosphatase measurement.