SNCA and tauopathy: Moreover, oligodendroglial PrPd deposits were comparable with coiled bodies observed in the four-repeat tauopathies, such as progressive supranuclear palsy, corticobasal degeneration or argyrophilic grain disease, or even glial cytoplasmic alpha-synuclein inclusions of MSA, as some PrPd aggregates appeared coarser or microglobular.