Compared to patients without AKI, those patients who developed AKI had a significantly higher median suPAR (5.8 ng/mL vs. 4.8 ng/mL, p < 0.001 (Figure 1a)) and higher median NGAL (229 ng/mL vs. 105 ng/mL, p < 0.001 (Figure 1b). The gene discussed is LCN2; the disease is acute kidney injury.